By J. E. Fischer M.D. (auth.), J. M. Greep, Peter B. Soeters, R. I. C. Wesdorp, C. W. R. Phaf, Josef E. Fischer M.D. (eds.)
J. E. Fischer, M.D. Professor Greep, women and gents. it's a nice excitement for me and the members to be current at this overseas assembly of Parenteral Nutri tion. This assembly wouldn't have been attainable 5 years in the past. at the moment we have been nonetheless arguing approximately significant vs. peripheral routes, efficacy of the tech nique, and nonetheless getting acquainted with our skill to aid sufferers nutri tionally. in the final 5 years those discussions, which appear virtually futile on reflection, were set aside. either suggestions, we all know how, paintings fairly good and feature their very own symptoms. Having turn into pleased with the strategy, it's now time to go into the second one part of parenteral food, and that's the differentiation of the procedure for the advantage of diverse sufferers. Over the following days we are going to be discussing a number of difficulties which today are imperative to the whole topic of parenteral foodstuff. Is a fats calorie kind of like a carbo hydrate calorie? what's the impact on protein metabolism of the fats calorie in preference to the carbohydrate calorie? Are they an identical? Are'there occasions during which one is better to the opposite? probably we'll discover the following day within the panel.
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E. their catabolic and anti-insulin properties, and to feed-back regulation disturbances. These factors will lead to a diabetic-like glucose intolerance and I resistance not only because of interference with glucose phosphorylation mechanisms but also because of the increase in FFA that they induce. The positive glucose effect on I and the negative one on G secretion are depressed. More glucose will be needed to produce them, so that the need for exogenous I is then better understood. + Hormonal control 49 Amino acids when administered alone will find a set of hormonal and enzymatic activities oriented mainly toward catabolism and gluconeogenesis.
In post-operative situations in which liver functions are already impaired by the use of narcotics, this may cause a dangerous situation. Moreover ethanol may give rise to problems as a result of its effect on the central nervous system, which in pre-existing hypoxia might be very dangerous. In view of the above-mentioned disadvantages we might conclude that ethanol should not be used as an energy source. 1 gjkg Bwjhr (51). Carbohydrates and polyalcohols as energy source After total metabolism, 100 g of such preparations can provide 400 Kcal.
5. Allison, S. , Metabolic aspects of intensive care. Br. J. Hosp. , 860-872 (1974). 6. Allison, S. , Carbohydrate and Fat Metabolism - Response to Injury. In: Lee, H. A. ), Parenteral Nutrition in Acute Metabolic Illness, London, 1974, pp. 167-175. 7. Bassler, K. H. and Reimold, W. , Lactatbildung aus Zuckern und Zuckeralkoholen in Erythrozyten. Klin. Wschr. 43: 169 (1965). 8. Bassler, K. , Stein, G. , Xylitstoffwechsel und Xylitresorption. Biochern Z. 346: 171 (1966). 9. Bassler, K. , Empfehlungen zur parenteralen Ernahrung.