By Professor Michael Forsting MD, PhD (auth.), Michael Forsting MD, PhD, Isabel Wanke MD, PhD (eds.)
This e-book describes the pathoanatomical, pathophysiological, and imaging beneficial properties of vascular malformations and aneurysms of the mind and the fashionable, minimally invasive endovascular equipment and strategies hired of their remedy. person chapters are dedicated to developmental venous malformations, cavernomas and capillary telangiectasias, pial arteriovenous malformations, dural arteriovenous malformations, and intracranial aneurysms. each one bankruptcy is subdivided into 4 critical sections on pathology, medical presentation, diagnostic imaging, and remedy, making sure a standardized strategy all through. All chapters during this second revised version of Intracranial Vascular Malformations and Aneurysms were completely up-to-date. The publication is richly illustrated with quite a few informative CT, MR and DSA photographs, together with high-end 7-Tesla MR images.
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Extra resources for Intracranial Vascular Malformations and Aneurysms: From Diagnostic Work-Up to Endovascular Therapy
A Computer tomography scan without intravenous contrast. The large, hyperdense lesion in the white matter adjacent to the lateral ventricle is not surrounded by edema, nor is there any blood in the ventricle itself. b In this window setting, calciﬁed areas of the wall and parts of the inner structures are clearly visible. c The T2-weighted FLAIR sequence shows a dark mass lesion protruding into the ventricle. There is no perifocal edema. d T1-weighted spin-echo sequence without contrast reveals the lesion as hyperintense.
Time-of-ﬂight MRA sequences are usually heavily T1-weighted. Therefore, a cavernoma can mimic an aneurysm on these images (Fig. 8). Most of the clinically used DWI sequences are T2*-weighted and, thus, should detect cavernomas with an increased sensitivity (see Fig. 11). Zabramski et al. 1). The problem of this classiﬁcation is the type-4 lesion. The pathologic deﬁnition of this type is totally unclear and for us it is questionable whether these lesions really represent capillary telangiectasias.
D Coronal T2-weighted image also demonstrates the hemosiderin deposition within the white matter and not on the surface of the brain. e This maximum-intensity projection of a time-of-ﬂight MRA shows a structure adjacent to the anterior cerebral arteries. This is due to the high T1 signal of the cavernoma, indistinguishable from the ﬂow signal in a T1-weighted FISP-MRA sequence. This phenomenon can be misinterpreted as an aneurysm. f Intraarterial digital subtraction angiography to rule out an aneurysm.